[1] ZUO T, XIE Q, LIU J F, et al. Macrophage-derived cathepsin S remodels the extracellular matrix to promote liver fibrogenesis[J]. Gastroenterology, 2023, 165(3): 746-761. e16.
[2] COLL M, ARIÑO S, MARTÍNEZ-SÁNCHEZ C, et al. Ductular reaction promotes intrahepatic angiogenesis through Slit2-Round about 1 signaling[J]. Hepatology, 2022, 75(2):353-368. doi:  10.1002/hep.32140
[3] CHOUDHURY A, RATNA A, LIM A, et al. Loss of heat shock factor 1 promotes hepatic stellate cell activation and drives liver fibrosis[J]. Hepatol Commun, 2022, 6(10):2781-2797. doi:  10.1002/hep4.2058
[4] YU X J, ELFIMOVA N, MÜLLER M, et al. Autophagy-related activation of hepatic stellate cells reduces cellular miR-29a by promoting its vesicular secretion[J]. Cell Mol Gastroenterol Hepatol, 2022, 13(6):1701-1716. doi:  10.1016/j.jcmgh.2022.02.013
[5] WU S S, LI S F, JIN P, et al. Interplay between hypertriglyceridemia and acute promyelocytic leukemia mediated by the cooperation of peroxisome proliferator-activated receptor-α with the PML/RAR α fusion protein on super-enhancers[J]. Haematologica, 2022, 107(11):2589-2600. doi:  10.3324/haematol.2021.280147
[6] SHIMIZU H, TSUBOTA T, KANKI K, et al. All-trans retinoic acid ameliorates hepatic stellate cell activation via suppression of thioredoxin interacting protein expression[J]. J Cell Physiol, 2018, 233(1):607-616. doi:  10.1002/jcp.25921
[7] ROCKEY D C. Liver fibrosis reversion after suppression of hepatitis B virus[J]. Clin Liver Dis, 2016, 20(4):667-679. doi:  10.1016/j.cld.2016.06.003
[8] DE MESQUITA F C, GUIXÉ-MUNTET S, FERNÁNDEZ-IGLESIAS A, et al. Liraglutide improves liver microvascular dysfunction in cirrhosis: evidence from translational studies[J]. Sci Rep, 2017, 7(1):3255. doi:  10.1038/s41598-017-02866-y
[9] LEE Y A, WALLACE M C, FRIEDMAN S L. Pathobiology of liver fibrosis: a translational success story[J]. Gut, 2015, 64(5):830-841. doi:  10.1136/gutjnl-2014-306842
[10] SCORLETTI E, CARR R M. A new perspective on NAFLD: focusing on lipid droplets[J]. J Hepatol, 2022, 76(4):934-945. doi:  10.1016/j.jhep.2021.11.009
[11] KLUWE J, WONGSIRIROJ N, TROEGER J S, et al. Absence of hepatic stellate cell retinoid lipid droplets does not enhance hepatic fibrosis but decreases hepatic carcinogenesis[J]. Gut, 2011, 60(9):1260-1268. doi:  10.1136/gut.2010.209551
[12] SENOO H, MEZAKI Y, FUJIWARA M. The stellate cell system(vitamin A-storing cell system)[J]. Anat Sci Int, 2017, 92(4):387-455. doi:  10.1007/s12565-017-0395-9
[13] LI X Y, LUO X Q, CHEN S R, et al. All-trans-retinoic acid inhibits hepatocellular carcinoma progression by targeting myeloid-derived suppressor cells and inhibiting angiogenesis[J]. Int Immunopharmacol, 2023, 121:110413. doi:  10.1016/j.intimp.2023.110413
[14] XIA S L, LIU Z M, CAI J R, et al. Liver fibrosis therapy based on biomimetic nanoparticles which deplete activated hepatic stellate cells[J]. J Control Release, 2023, 355:54-67. doi:  10.1016/j.jconrel.2023.01.052
[15] SCHUG T T, BERRY D C, SHAW N S, et al. Opposing effects of retinoic acid on cell growth result from alternate activation of two different nuclear receptors[J]. Cell, 2007, 129(4):723-733. doi:  10.1016/j.cell.2007.02.050
[16] PANEBIANCO C, OBEN J A, VINCIGUERRA M, et al. Senescence in hepatic stellate cells as a mechanism of liver fibrosis reversal: a putative synergy between retinoic acid and PPAR-gamma signalings[J]. Clin Exp Med, 2017, 17(3):269-280. doi:  10.1007/s10238-016-0438-x
[17] RUART M, CHAVARRIA L, CAMPRECIÓS G, et al. Impaired endothelial autophagy promotes liver fibrosis by aggravating the oxidative stress response during acute liver injury[J]. J Hepatol, 2019, 70(3):458-469. doi:  10.1016/j.jhep.2018.10.015
[18] LUANGMONKONG T, SURIGUGA S, MUTSAERS H A M, et al. Targeting oxidative stress for the treatment of liver fibrosis[J]. Rev Physiol Biochem Pharmacol, 2018, 175:71-102.
[19] YANG L, BI L P, JIN L L, et al. Geniposide ameliorates liver fibrosis through reducing oxidative stress and inflammatory respose, inhibiting apoptosis and modulating overall metabolism[J]. Front Pharmacol, 2021, 12:772635. doi:  10.3389/fphar.2021.772635
[20] NARAYANANKUTTY A, JOB J T, NARAYANANKUTTY V. Glutathione, an antioxidant tripeptide: dual roles in carcinogenesis and chemoprevention[J]. Curr Protein Pept Sci, 2019, 20(9):907-917. doi:  10.2174/1389203720666190206130003
[21] GU J Y, CHEN C, WANG J, et al. Withaferin A exerts preventive effect on liver fibrosis through oxidative stress inhibition in a sirtuin 3-dependent manner[J]. Oxid Med Cell Longev, 2020, 2020:2452848.
[22] HAZARI Y, BRAVO-SAN PEDRO J M, HETZ C, et al. Autophagy in hepatic adaptation to stress[J]. J Hepatol, 2020, 72(1):183-196. doi:  10.1016/j.jhep.2019.08.026
[23] UENO T, KOMATSU M. Autophagy in the liver: functions in health and disease[J]. Nat Rev Gastroenterol Hepatol, 2017, 14(3):170-184. doi:  10.1038/nrgastro.2016.185
[24] HERNÁNDEZ-GEA V, FRIEDMAN S L. Autophagy fuels tissue fibrogenesis[J]. Autophagy, 2012, 8(5):849-850. doi:  10.4161/auto.19947
[25] HERNÁNDEZ-GEA V, GHIASSI-NEJAD Z, ROZENFELD R, et al. Autophagy releases lipid that promotes fibrogenesis by activated hepatic stellate cells in mice and in human tissues[J]. Gastroenterology, 2012, 142(4):938-946. doi:  10.1053/j.gastro.2011.12.044
[26] SEKI E, BRENNER D A. Recent advancement of molecular mechanisms of liver fibrosis[J]. J Hepatobiliary Pancreat Sci, 2015, 22(7):512-518. doi:  10.1002/jhbp.245
[27] ZAHMATKESH E, OTHMAN A, BRAUN B, et al. In vitro modeling of liver fibrosis in 3D microtissues using scalable micropatterning system[J]. Arch Toxicol, 2022, 96(6):1799-1813. doi:  10.1007/s00204-022-03265-7
[28] ASRANI S K, DEVARBHAVI H, EATON J, et al. Burden of liver diseases in the world[J]. J Hepatol, 2019, 70(1):151-171. doi:  10.1016/j.jhep.2018.09.014
[29] LUCANTONI F, MARTÍNEZ-CEREZUELA A, GRUEVSKA A, et al. Understanding the implication of autophagy in the activation of hepatic stellate cells in liver fibrosis: are we there yet?[J]. J Pathol, 2021, 254(3):216-228. doi:  10.1002/path.5678
[30] XU L, HUI A Y, ALBANIS E, et al. Human hepatic stellate cell lines, LX-1 and LX-2: new tools for analysis of hepatic fibrosis[J]. Gut, 2005, 54(1):142-151. doi:  10.1136/gut.2004.042127